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Madopar monotherapy

Peking University Third Hospital · FDA-approved active Small molecule

Madopar combines levodopa and benserazide to increase dopamine levels in the brain by delivering levodopa across the blood-brain barrier while benserazide blocks its peripheral conversion.

Madopar is a combination of levodopa and benserazide that increases dopamine levels in the brain by providing the dopamine precursor while inhibiting its peripheral metabolism. Used for Parkinson's disease.

At a glance

Generic nameMadopar monotherapy
SponsorPeking University Third Hospital
Drug classDopamine precursor / Decarboxylase inhibitor combination
TargetDopamine pathway; aromatic L-amino acid decarboxylase (peripheral inhibition by benserazide)
ModalitySmall molecule
Therapeutic areaNeurology
PhaseFDA-approved

Mechanism of action

Levodopa is a dopamine precursor that crosses the blood-brain barrier and is converted to dopamine in the central nervous system. Benserazide is a peripheral decarboxylase inhibitor that prevents levodopa from being converted to dopamine outside the brain, thereby increasing the amount of levodopa available for central conversion and reducing peripheral side effects. This combination allows lower doses of levodopa to be effective while minimizing gastrointestinal and cardiovascular adverse effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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