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Low dose ASA

University of Pennsylvania · Phase 3 active Small molecule

Low-dose aspirin inhibits platelet cyclooxygenase to reduce thromboxane A2 production, preventing platelet aggregation and clot formation.

Low-dose aspirin inhibits platelet cyclooxygenase to reduce thromboxane A2 production, preventing platelet aggregation and clot formation. Used for Primary prevention of cardiovascular events, Secondary prevention of myocardial infarction and stroke, Atrial fibrillation for stroke prevention.

At a glance

Generic nameLow dose ASA
Also known asLow-dose aspirin
SponsorUniversity of Pennsylvania
Drug classAntiplatelet agent / NSAID
TargetCyclooxygenase-1 (COX-1)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

Aspirin irreversibly acetylates cyclooxygenase-1 (COX-1) in platelets, blocking thromboxane A2 synthesis. This reduces platelet activation and aggregation, thereby decreasing thrombotic events. Low-dose formulations (typically 75–325 mg daily) preferentially affect platelet function while minimizing gastrointestinal side effects associated with higher doses.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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