Last reviewed · How we verify

Levodopa/carbidopa/entacapone

Novartis · FDA-approved active Small molecule

Levodopa is converted to dopamine in the brain to restore dopamine levels, while carbidopa blocks its peripheral conversion and entacapone inhibits catechol-O-methyltransferase to extend levodopa's duration of action.

Levodopa is converted to dopamine in the brain to restore dopamine levels, while carbidopa blocks its peripheral conversion and entacapone inhibits catechol-O-methyltransferase to extend levodopa's duration of action. Used for Parkinson's disease with motor fluctuations.

At a glance

Generic nameLevodopa/carbidopa/entacapone
Also known asStalevo
SponsorNovartis
Drug classDopamine precursor with decarboxylase inhibitor and COMT inhibitor
TargetDopamine pathway; COMT enzyme
ModalitySmall molecule
Therapeutic areaNeurology
PhaseFDA-approved

Mechanism of action

Levodopa crosses the blood-brain barrier and is converted to dopamine, the neurotransmitter deficient in Parkinson's disease. Carbidopa is a decarboxylase inhibitor that prevents levodopa's conversion to dopamine in the periphery, allowing more to reach the brain and reducing side effects. Entacapone inhibits COMT, an enzyme that metabolizes dopamine, thereby prolonging levodopa's therapeutic effect and reducing motor fluctuations.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: