Last reviewed · How we verify

JAK Inhibitor

Fred Hutchinson Cancer Center · FDA-approved active Small molecule

JAK inhibitors block Janus kinase enzymes, which are intracellular signaling proteins that transmit inflammatory and immune signals from cell surface receptors.

JAK inhibitors block Janus kinase enzymes, which are intracellular signaling proteins that transmit inflammatory and immune signals from cell surface receptors. Used for Myeloproliferative neoplasms (e.g., polycythemia vera, essential thrombocythemia, primary myelofibrosis), Hematologic malignancies and related conditions.

At a glance

Generic nameJAK Inhibitor
Also known asJAK inhibitors, Janus Kinase Inhibitor, Ruxolitinib, Fedratinib, Tofacitinib, baricitinib, or upadacitinib
SponsorFred Hutchinson Cancer Center
Drug classJAK inhibitor
TargetJAK1, JAK2, JAK3, and/or TYK2
ModalitySmall molecule
Therapeutic areaOncology, Immunology
PhaseFDA-approved

Mechanism of action

JAK inhibitors work by inhibiting one or more of the four JAK family kinases (JAK1, JAK2, JAK3, TYK2), thereby suppressing the JAK-STAT signaling pathway. This pathway is critical for cytokine and growth factor signaling in immune and hematopoietic cells. By blocking JAK activity, these inhibitors reduce inflammatory cytokine production and immune cell activation, making them useful in both hematologic malignancies and inflammatory/autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: