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Ivacaftor (VX-770)

University of Alabama at Birmingham · FDA-approved active Small molecule

Ivacaftor is a CFTR potentiator that increases the open probability and activity of the cystic fibrosis transmembrane conductance regulator (CFTR) protein at the cell surface.

Ivacaftor is a CFTR potentiator that increases the open probability and activity of the cystic fibrosis transmembrane conductance regulator (CFTR) protein at the cell surface. Used for Cystic fibrosis in patients with G551D mutation in CFTR gene, Cystic fibrosis in patients with other gating mutations (R117H, R334W, R347P, R352Q, A455E, S549N, S549R, G1244E, S1251N, S1255P, S1255R, G1349D), Cystic fibrosis in patients with F508del-CFTR mutation (in combination with lumacaftor or tezacaftor).

At a glance

Generic nameIvacaftor (VX-770)
SponsorUniversity of Alabama at Birmingham
Drug classCFTR potentiator
TargetCFTR (cystic fibrosis transmembrane conductance regulator)
ModalitySmall molecule
Therapeutic areaRespiratory / Genetic Disorders
PhaseFDA-approved

Mechanism of action

Ivacaftor binds directly to the CFTR protein and enhances its channel-opening activity, allowing increased chloride ion transport across epithelial cell membranes. This mechanism is particularly effective for CFTR mutations that produce protein that reaches the cell surface but has reduced function (gating mutations). By restoring CFTR function, ivacaftor improves mucociliary clearance and reduces airway inflammation in cystic fibrosis patients.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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