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IV N-acetylcysteine

Medical University of South Carolina · Phase 3 active Small molecule

IV N-acetylcysteine replenishes intracellular glutathione and acts as a free radical scavenger to reduce oxidative stress and inflammation.

IV N-acetylcysteine replenishes intracellular glutathione and acts as a free radical scavenger to reduce oxidative stress and inflammation. Used for Acetaminophen (paracetamol) overdose, Acute respiratory distress syndrome (ARDS), Chronic obstructive pulmonary disease (COPD) exacerbations.

At a glance

Generic nameIV N-acetylcysteine
Also known asNAC
SponsorMedical University of South Carolina
Drug classAntioxidant / Mucolytic agent
TargetGlutathione synthesis pathway; free radical scavenger
ModalitySmall molecule
Therapeutic areaCritical Care / Toxicology / Respiratory
PhasePhase 3

Mechanism of action

N-acetylcysteine (NAC) is a prodrug that is rapidly deacetylated to cysteine, which serves as a precursor for glutathione synthesis. By increasing intracellular glutathione levels, NAC enhances antioxidant capacity and reduces reactive oxygen species (ROS). Additionally, NAC directly scavenges free radicals and modulates inflammatory pathways, making it useful in conditions characterized by oxidative stress and tissue damage.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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