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Isoniazid (INH)

National Institutes of Health Clinical Center (CC) · FDA-approved active Small molecule

Isoniazid inhibits mycobacterial cell wall synthesis by interfering with mycolic acid production, thereby killing Mycobacterium tuberculosis.

Isoniazid inhibits mycobacterial cell wall synthesis by interfering with mycolic acid production, thereby killing Mycobacterium tuberculosis. Used for Tuberculosis (active disease), Tuberculosis prophylaxis (latent TB infection).

At a glance

Generic nameIsoniazid (INH)
Also known asGeneric (various), isonicotinyl hydrazine,
SponsorNational Institutes of Health Clinical Center (CC)
Drug classAntituberculous agent
TargetInhA (enoyl-ACP reductase)
ModalitySmall molecule
Therapeutic areaInfectious Disease
PhaseFDA-approved

Mechanism of action

Isoniazid is a prodrug that requires activation by the mycobacterial enzyme catalase-peroxidase (KatG) to form an isonicotinoyl radical, which then inhibits InhA, an enoyl-ACP reductase essential for mycolic acid synthesis. Mycolic acids are critical components of the mycobacterial cell wall, and their disruption leads to cell wall instability and bacterial death. This mechanism is selective for mycobacteria, making it highly effective against tuberculosis.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results