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intravitreal ranibizumab & photodynamic therapy

University Hospital, Basel, Switzerland · Phase 3 active Small molecule

Intravitreal ranibizumab inhibits vascular endothelial growth factor A (VEGF-A) by binding to its receptor, reducing angiogenesis and edema in the retina.

Intravitreal ranibizumab inhibits vascular endothelial growth factor A (VEGF-A) by binding to its receptor, reducing angiogenesis and edema in the retina. Used for Treatment of neovascular (wet) age-related macular degeneration, Treatment of macular edema following branch retinal vein occlusion or central retinal vein occlusion.

At a glance

Generic nameintravitreal ranibizumab & photodynamic therapy
Also known asVisudyne
SponsorUniversity Hospital, Basel, Switzerland
Drug classAnti-VEGF agent
TargetVEGF-A
ModalitySmall molecule
Therapeutic areaOphthalmology
PhasePhase 3

Mechanism of action

Ranibizumab is a recombinant humanized monoclonal antibody fragment that targets VEGF-A, a key mediator of pathological angiogenesis. By binding to VEGF-A, ranibizumab prevents its interaction with its receptor, thereby inhibiting the downstream signaling pathways that lead to angiogenesis and edema. Photodynamic therapy involves the use of a light-sensitive medication and a low-power laser to activate the medication and produce a chemical reaction that destroys abnormal blood vessels in the retina.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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