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Intravenous rhTNK-tPA

Xinqiao Hospital of Chongqing · Phase 3 active Small molecule

rhTNK-tPA is a recombinant thrombolytic agent that dissolves blood clots by converting plasminogen to plasmin, enabling fibrin degradation.

rhTNK-tPA is a recombinant thrombolytic agent that dissolves blood clots by converting plasminogen to plasmin, enabling fibrin degradation. Used for Acute ischemic stroke, Acute myocardial infarction.

At a glance

Generic nameIntravenous rhTNK-tPA
Also known asIntravenous tenecteplase, Intravenous TNK
SponsorXinqiao Hospital of Chongqing
Drug classThrombolytic agent / Fibrinolytic
TargetPlasminogen / Fibrin
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

This drug is a genetically engineered tissue plasminogen activator (tPA) variant designed to improve thrombolytic efficacy and safety in acute thrombotic conditions. By enhancing fibrin specificity and reducing systemic plasminogen activation compared to conventional tPA, it aims to achieve faster clot dissolution with potentially lower bleeding risk. The recombinant human TNK-tPA (tenecteplase-like variant) is administered intravenously for rapid thrombus resolution.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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