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intravenous levetireacetam

ayesha tariq · FDA-approved active Small molecule

Levetiracetam reduces neuronal excitability and seizure activity through binding to synaptic vesicle protein SV2A, modulating neurotransmitter release.

Levetiracetam reduces neuronal excitability and seizure activity through binding to synaptic vesicle protein SV2A, modulating neurotransmitter release. Used for Adjunctive therapy for partial-onset seizures, Adjunctive therapy for myoclonic seizures in patients with juvenile myoclonic epilepsy, Adjunctive therapy for primary generalized tonic-clonic seizures.

At a glance

Generic nameintravenous levetireacetam
Sponsorayesha tariq
Drug classAntiepileptic agent
TargetSV2A (synaptic vesicle protein 2A)
ModalitySmall molecule
Therapeutic areaNeurology
PhaseFDA-approved

Mechanism of action

Levetiracetam is a pyrrolidone derivative that binds to the synaptic vesicle protein SV2A, which is involved in vesicle exocytosis and neurotransmitter release. This binding reduces the release of excitatory neurotransmitters and stabilizes neuronal membranes, thereby suppressing seizure propagation. The exact mechanism remains incompletely understood, but SV2A binding is considered the primary molecular action responsible for its anticonvulsant effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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