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Interferon Alfa、Fluorouracil

Kansai Hepatobiliary Oncology Group · Phase 2 active Small molecule

Interferon alfa binds to interferon receptors on the surface of cells, leading to the activation of JAK-STAT signaling pathways which enhance antiviral and antiproliferative responses; Fluorouracil inhibits thymidylate synthase, disrupting DNA synthesis and repair in rapidly dividing cells.

Interferon alfa binds to interferon receptors on the surface of cells, leading to the activation of JAK-STAT signaling pathways which enhance antiviral and antiproliferative responses; Fluorouracil inhibits thymidylate synthase, disrupting DNA synthesis and repair in rapidly dividing cells. Used for Hepatocellular carcinoma.

At a glance

Generic nameInterferon Alfa、Fluorouracil
Also known asInterferon Alfa ; IFN、, Fluorouracil ; 5-FU
SponsorKansai Hepatobiliary Oncology Group
Drug classInterferon alfa: cytokine; Fluorouracil: antimetabolite
TargetInterferon alfa: IFNAR1/IFNAR2; Fluorouracil: thymidylate synthase
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 2

Mechanism of action

Interferon alfa activates the JAK-STAT pathway, which increases the expression of genes involved in antiviral defense and immune modulation. Fluorouracil's inhibition of thymidylate synthase leads to a depletion of thymidine triphosphate, essential for DNA replication, thereby inducing apoptosis in cancer cells.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results