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Inqovi

Otsuka · FDA-approved active Small molecule ✓ Verified May 2026 Quality 48/100

Inqovi is a Nucleoside Metabolic Inhibitor [EPC] Small molecule drug developed by Otsuka. It is currently FDA-approved (first approved 2020) for Chronic myelomonocytic leukemia, Myelodysplastic syndrome. Also known as: Decitabine, Cedazuridine, Decitabine and Cedazuridine, ASTX727.

Inqovi works by blocking the enzyme cytidine deaminase, allowing chemotherapy medications to be more effective.

Inqovi is a fixed-dose combination anticancer medication used for the treatment of adults with myelodysplastic syndromes and chronic myelomonocytic leukemia (CMML). It is a combination of decitabine, a nucleoside metabolic inhibitor, and cedazuridine, a cytidine deaminase inhibitor.

At a glance

Generic nameInqovi
Also known asDecitabine, Cedazuridine, Decitabine and Cedazuridine, ASTX727, decitabine
SponsorOtsuka
Drug classNucleoside Metabolic Inhibitor [EPC]
TargetCytidine deaminase
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved
First approval2020

Mechanism of action

Decitabine is nucleoside metabolic inhibitor that is believed to exert its effects after phosphorylation and direct incorporation into DNA and inhibition of DNA methyltransferase, causing hypomethylation of DNA and cellular differentiation and/or apoptosis. Decitabine inhibits DNA methylation in vitro, which is achieved at concentrations that do not cause major suppression of DNA synthesis. Decitabine-induced hypomethylation in cancer cells may restore normal function to genes that are critical for the control of cellular differentiation and proliferation. In rapidly dividing cells, the cytotoxicity of decitabine may also be attributed to the formation of covalent adducts between DNA methyltransferase and decitabine incorporated into DNA. Non-proliferating cells are relatively insensitive to decitabine.Cytidine deaminase (CDA) is an enzyme that catalyzes the degradation of cytidine, including the cytidine analog decitabine. High levels of CDA in the gastrointe

Approved indications

Common side effects

Key clinical trials

Patents

PatentExpiryType

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results
FDA Orange BookPatents + exclusivity

Competitive intelligence

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Frequently asked questions about Inqovi

What is Inqovi?

Inqovi is a Nucleoside Metabolic Inhibitor [EPC] drug developed by Otsuka, indicated for Chronic myelomonocytic leukemia, Myelodysplastic syndrome.

How does Inqovi work?

Inqovi works by blocking the enzyme cytidine deaminase, allowing chemotherapy medications to be more effective.

What is Inqovi used for?

Inqovi is indicated for Chronic myelomonocytic leukemia, Myelodysplastic syndrome.

Who makes Inqovi?

Inqovi is developed and marketed by Otsuka (see full Otsuka pipeline at /company/otsuka).

Is Inqovi also known as anything else?

Inqovi is also known as Decitabine, Cedazuridine, Decitabine and Cedazuridine, ASTX727, decitabine.

What drug class is Inqovi in?

Inqovi belongs to the Nucleoside Metabolic Inhibitor [EPC] class. See all Nucleoside Metabolic Inhibitor [EPC] drugs at /class/nucleoside-metabolic-inhibitor-epc.

When was Inqovi approved?

Inqovi was first approved on 2020.

What development phase is Inqovi in?

Inqovi is FDA-approved (marketed).

What are the side effects of Inqovi?

Common side effects of Inqovi include Fatigue, Hemorrhage, Edema, Pyrexia, Constipation, Mucositis.

What does Inqovi target?

Inqovi targets Cytidine deaminase and is a Nucleoside Metabolic Inhibitor [EPC].

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing