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Remicade (infliximab)

Johnson & Johnson · discontinued Monoclonal antibody Quality 43/100

Infliximab binds to TNFα, blocking its interaction with receptors, reducing inflammation and immune responses.

Infliximab (Remicade), developed by Johnson & Johnson, holds a strong market position as one of the leading anti-TNFα therapies with 9 approved indications. Its competitive advantage lies in its well-established mechanism of action, which effectively reduces inflammation and immune responses by binding to TNFα. A key risk is the lack of ongoing clinical trials, which may limit future expansion and innovation. The pipeline outlook remains uncertain, particularly given the presence of strong competitors like etanercept, adalimumab, certolizumab pegol, and golimumab, and the requirement for a PD-L1 companion diagnostic for several indications.

At a glance

Generic nameinfliximab
SponsorJohnson & Johnson
Drug classMonoclonal antibody
TargetTNFα
ModalityMonoclonal antibody
Therapeutic areaOncology
Phasediscontinued
First approval1998

Mechanism of action

Infliximab works by binding to both soluble and transmembrane forms of TNFα, a protein that plays a key role in inflammation. By blocking TNFα, infliximab prevents it from binding to its receptors, thereby reducing the production of pro-inflammatory cytokines and other inflammatory responses. This helps to alleviate symptoms in conditions like rheumatoid arthritis and inflammatory bowel disease.

Approved indications

Common side effects

Serious adverse events

Drug interactions

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions

Competitive intelligence

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