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IL-23 Therapy

Weill Medical College of Cornell University · Phase 3 active Biologic

IL-23 therapy inhibits the IL-23 signaling pathway to reduce pathogenic T-cell responses and inflammatory cytokine production.

IL-23 therapy inhibits the IL-23 signaling pathway to reduce pathogenic T-cell responses and inflammatory cytokine production. Used for Psoriasis, Inflammatory bowel disease (Crohn's disease and ulcerative colitis), Psoriatic arthritis.

At a glance

Generic nameIL-23 Therapy
Also known asRisankizumab, Ustekinumab, Guselkumab
SponsorWeill Medical College of Cornell University
Drug classIL-23 inhibitor
TargetIL-23 (interleukin-23)
ModalityBiologic
Therapeutic areaImmunology
PhasePhase 3

Mechanism of action

IL-23 is a key cytokine that drives Th17 cell differentiation and maintains inflammatory responses in autoimmune and inflammatory diseases. By blocking IL-23 or its receptor, this therapy reduces the expansion and activation of pro-inflammatory T cells, thereby suppressing disease-driving immune responses. This mechanism has shown efficacy in conditions characterized by IL-23-mediated inflammation, particularly in skin and gut-associated diseases.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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