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Ibrutinib combined with As2O3

Peking University People's Hospital · Phase 3 active Small molecule

Ibrutinib inhibits Bruton's tyrosine kinase (BTK) to block B-cell proliferation, while arsenic trioxide (As2O3) induces apoptosis and differentiation in leukemic cells through multiple pathways.

Ibrutinib inhibits Bruton's tyrosine kinase (BTK) to block B-cell proliferation, while arsenic trioxide (As2O3) induces apoptosis and differentiation in leukemic cells through multiple pathways. Used for Acute leukemia (investigational combination therapy), B-cell malignancies with arsenic sensitization.

At a glance

Generic nameIbrutinib combined with As2O3
Also known asIbrutinib combined with arsenic trioxide
SponsorPeking University People's Hospital
Drug classBTK inhibitor combined with arsenic compound
TargetBTK (Bruton's tyrosine kinase); As2O3 has multiple targets including PML-RARA fusion protein and mitochondrial pathways
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Ibrutinib is a BTK inhibitor that disrupts B-cell receptor signaling, preventing malignant B-cell survival and proliferation. Arsenic trioxide acts as a dual-mechanism agent that promotes apoptosis and differentiation of leukemic blasts, particularly in acute promyelocytic leukemia. The combination leverages complementary mechanisms to enhance anti-leukemic activity.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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