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ibandronate, calcium and vitamin D

Charite University, Berlin, Germany · FDA-approved active Small molecule

Ibandronate inhibits osteoclast-mediated bone resorption by binding to hydroxyapatite in bone and blocking farnesyl pyrophosphate synthase, while calcium and vitamin D support bone mineralization and calcium homeostasis.

Ibandronate inhibits osteoclast-mediated bone resorption by binding to hydroxyapatite in bone and blocking farnesyl pyrophosphate synthase, while calcium and vitamin D support bone mineralization and calcium homeostasis. Used for Osteoporosis prevention and treatment in postmenopausal women, Bone loss associated with glucocorticoid therapy.

At a glance

Generic nameibandronate, calcium and vitamin D
SponsorCharite University, Berlin, Germany
Drug classBisphosphonate combination therapy
TargetFarnesyl pyrophosphate synthase (ibandronate); calcium-sensing receptor and vitamin D receptor (supportive agents)
ModalitySmall molecule
Therapeutic areaBone metabolism / Osteoporosis
PhaseFDA-approved

Mechanism of action

Ibandronate is a nitrogen-containing bisphosphonate that reduces bone turnover by inhibiting osteoclast activity and promoting osteoclast apoptosis. Calcium provides the mineral substrate for bone formation, and vitamin D enhances intestinal calcium absorption and regulates calcium-phosphate metabolism, together reducing fracture risk in osteoporosis.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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