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i.v. selexipag

Actelion · Phase 3 active Small molecule Under review

i.v. selexipag is a prostacyclin receptor agonist Small molecule drug developed by Actelion. It is currently in Phase 3 development for Pulmonary arterial hypertension (PAH). Also known as: ACT-293987.

Selexipag is a prostacyclin receptor agonist that acts as a potent vasodilator.

Selexipag is a small molecule that acts as a prostanoid IP receptor agonist. It is used to treat Pulmonary Arterial Hypertension, a condition where the blood pressure in the lungs is elevated.

Likelihood of approval
56.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Cardiovascular Phase 3 risk -2.0pp
    Modern cardiovascular outcome trials are large + long; many fail to beat aggressive standard-of-care.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic namei.v. selexipag
Also known asACT-293987
SponsorActelion
Drug classprostacyclin receptor agonist
TargetIP receptor
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

Selexipag works by binding to the prostacyclin receptor, leading to increased levels of cyclic AMP in vascular smooth muscle cells, resulting in vasodilation and improved blood flow. This mechanism is particularly beneficial for patients with pulmonary arterial hypertension (PAH).

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about i.v. selexipag

What is i.v. selexipag?

i.v. selexipag is a prostacyclin receptor agonist drug developed by Actelion, indicated for Pulmonary arterial hypertension (PAH).

How does i.v. selexipag work?

Selexipag is a prostacyclin receptor agonist that acts as a potent vasodilator.

What is i.v. selexipag used for?

i.v. selexipag is indicated for Pulmonary arterial hypertension (PAH).

Who makes i.v. selexipag?

i.v. selexipag is developed by Actelion (see full Actelion pipeline at /company/actelion).

Is i.v. selexipag also known as anything else?

i.v. selexipag is also known as ACT-293987.

What drug class is i.v. selexipag in?

i.v. selexipag belongs to the prostacyclin receptor agonist class. See all prostacyclin receptor agonist drugs at /class/prostacyclin-receptor-agonist.

What development phase is i.v. selexipag in?

i.v. selexipag is in Phase 3.

What are the side effects of i.v. selexipag?

Common side effects of i.v. selexipag include Headache, Nausea, Dizziness.

What does i.v. selexipag target?

i.v. selexipag targets IP receptor and is a prostacyclin receptor agonist.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing