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High intensity statin monotherapy

Korea University Anam Hospital · FDA-approved active Small molecule

High-intensity statin monotherapy reduces LDL cholesterol by inhibiting HMG-CoA reductase, the rate-limiting enzyme in hepatic cholesterol synthesis.

High-intensity statin monotherapy reduces LDL cholesterol by inhibiting HMG-CoA reductase, the rate-limiting enzyme in hepatic cholesterol synthesis. Used for Primary prevention of atherosclerotic cardiovascular disease in high-risk patients, Secondary prevention following acute coronary syndrome or established coronary artery disease, Reduction of LDL cholesterol in hyperlipidemia.

At a glance

Generic nameHigh intensity statin monotherapy
SponsorKorea University Anam Hospital
Drug classHMG-CoA reductase inhibitor (statin)
TargetHMG-CoA reductase
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Statins competitively inhibit HMG-CoA reductase, blocking the conversion of HMG-CoA to mevalonate in the cholesterol biosynthetic pathway. This leads to decreased intracellular cholesterol levels, upregulation of LDL receptors on hepatocytes, and increased clearance of LDL particles from the bloodstream. High-intensity regimens (e.g., atorvastatin 40–80 mg or rosuvastatin 20–40 mg daily) achieve ≥50% LDL reduction and are used for primary and secondary cardiovascular prevention.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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