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High-intensity statin

Jung-min Ahn · FDA-approved active Small molecule

High-intensity statins inhibit HMG-CoA reductase to potently reduce LDL cholesterol synthesis in the liver.

High-intensity statins inhibit HMG-CoA reductase to potently reduce LDL cholesterol synthesis in the liver. Used for Primary prevention of cardiovascular disease in high-risk patients, Secondary prevention of cardiovascular events in patients with established coronary artery disease, Hypercholesterolemia.

At a glance

Generic nameHigh-intensity statin
SponsorJung-min Ahn
Drug classHMG-CoA reductase inhibitor (statin)
TargetHMG-CoA reductase
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Statins competitively inhibit 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase, the rate-limiting enzyme in cholesterol biosynthesis. This leads to decreased intracellular cholesterol levels, upregulation of LDL receptors on hepatocytes, and increased clearance of LDL cholesterol from the bloodstream. High-intensity statins (such as atorvastatin and rosuvastatin) achieve greater LDL reduction compared to moderate-intensity agents.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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