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Gonadotropin-releasing Hormone Analog

Rutgers, The State University of New Jersey · FDA-approved active Biologic

GnRH analogs bind to gonadotropin-releasing hormone receptors in the pituitary gland to suppress the release of luteinizing hormone and follicle-stimulating hormone, thereby reducing sex hormone production.

GnRH analogs bind to gonadotropin-releasing hormone receptors in the pituitary gland to suppress the release of luteinizing hormone and follicle-stimulating hormone, thereby reducing sex hormone production. Used for Advanced prostate cancer, Breast cancer (hormone-receptor positive), Endometriosis.

At a glance

Generic nameGonadotropin-releasing Hormone Analog
Also known asGnRH Agonist, GnRH Analog, Gonadotropin-Releasing Hormone Agonist, Gonadotropin-Releasing Hormone Analogue, LH-RH agonist
SponsorRutgers, The State University of New Jersey
Drug classGnRH agonist
TargetGnRH receptor
ModalityBiologic
Therapeutic areaOncology; Endocrinology
PhaseFDA-approved

Mechanism of action

GnRH analogs are synthetic peptides that mimic the natural GnRH hormone. Initially, they stimulate the pituitary gland, but with continuous exposure they cause desensitization and downregulation of GnRH receptors, leading to sustained suppression of gonadotropin secretion and consequent reduction in testosterone and estrogen levels. This mechanism is exploited therapeutically in hormone-dependent cancers and certain reproductive disorders.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results