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Glucocorticoid treatment

Prof Dr Frank Buttgereit · Phase 3 active Small molecule

Glucocorticoids suppress immune and inflammatory responses by binding to glucocorticoid receptors and modulating gene expression of pro-inflammatory cytokines and immune cells.

Glucocorticoids suppress immune and inflammatory responses by binding to glucocorticoid receptors and modulating gene expression of pro-inflammatory cytokines and immune cells. Used for Rheumatoid arthritis, Systemic lupus erythematosus, Polymyalgia rheumatica.

At a glance

Generic nameGlucocorticoid treatment
Also known asprednis(ol)one, methylprednisolone
SponsorProf Dr Frank Buttgereit
Drug classGlucocorticoid
TargetGlucocorticoid receptor (GR)
ModalitySmall molecule
Therapeutic areaImmunology, Rheumatology, Endocrinology
PhasePhase 3

Mechanism of action

Glucocorticoids are steroid hormones that enter cells and bind to intracellular glucocorticoid receptors, translocating to the nucleus to alter transcription of inflammatory genes. They inhibit production of cytokines (IL-1, IL-6, TNF-α), reduce immune cell activation and proliferation, and stabilize lysosomal membranes. This broad immunosuppressive and anti-inflammatory action makes them effective across multiple inflammatory and autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results