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GIK and intensive insulin therapy

University of Monastir · Phase 3 active Small molecule

GIK (glucose-insulin-potassium) therapy combined with intensive insulin regimens improves myocardial metabolism and reduces ischemic injury by enhancing glucose uptake and oxidative metabolism in cardiac tissue.

GIK (glucose-insulin-potassium) therapy combined with intensive insulin regimens improves myocardial metabolism and reduces ischemic injury by enhancing glucose uptake and oxidative metabolism in cardiac tissue. Used for Acute myocardial infarction with intensive glycemic control.

At a glance

Generic nameGIK and intensive insulin therapy
Also known asGIKI2
SponsorUniversity of Monastir
Drug classMetabolic therapy / Insulin-based combination
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

GIK therapy delivers a high-concentration glucose-insulin-potassium solution to shift myocardial metabolism toward glucose oxidation, which is more efficient under ischemic conditions than fatty acid oxidation. Intensive insulin therapy further optimizes glucose homeostasis and reduces inflammatory markers associated with acute coronary events. Together, these interventions aim to preserve cardiac function and reduce infarct size during acute myocardial infarction.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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