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fusidic acid [Verutex]

Hoffmann-La Roche · Phase 3 active Small molecule

fusidic acid [Verutex] is a Bacterial protein synthesis inhibitor Small molecule drug developed by Hoffmann-La Roche. It is currently in Phase 3 development for Bacterial infections, including osteomyelitis and septic arthritis.

Fusidic acid inhibits bacterial protein synthesis by binding to the elongation factor G.

Fusidic acid inhibits bacterial protein synthesis by binding to the elongation factor G. Used for Bacterial infections, including osteomyelitis and septic arthritis.

Likelihood of approval
63.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Anti-infectives pathway favourability +2.0pp
    Microbiological endpoints + non-inferiority designs raise approval rates above baseline.
  • Big-pharma sponsor +3.0pp
    Hoffmann-La Roche is a top-20 pharma sponsor — historical approval rates run ~3pp above average due to scale, regulatory experience, and trial-design quality.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic namefusidic acid [Verutex]
SponsorHoffmann-La Roche
Drug classBacterial protein synthesis inhibitor
TargetElongation factor G
ModalitySmall molecule
Therapeutic areaInfectious diseases
PhasePhase 3

Mechanism of action

This action prevents the translocation of peptidyl-tRNA from the A site to the P site on the ribosome, thereby inhibiting the elongation of the polypeptide chain. As a result, bacterial protein synthesis is halted, leading to cell death.

Approved indications

Common side effects

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about fusidic acid [Verutex]

What is fusidic acid [Verutex]?

fusidic acid [Verutex] is a Bacterial protein synthesis inhibitor drug developed by Hoffmann-La Roche, indicated for Bacterial infections, including osteomyelitis and septic arthritis.

How does fusidic acid [Verutex] work?

Fusidic acid inhibits bacterial protein synthesis by binding to the elongation factor G.

What is fusidic acid [Verutex] used for?

fusidic acid [Verutex] is indicated for Bacterial infections, including osteomyelitis and septic arthritis.

Who makes fusidic acid [Verutex]?

fusidic acid [Verutex] is developed by Hoffmann-La Roche (see full Hoffmann-La Roche pipeline at /company/roche).

What drug class is fusidic acid [Verutex] in?

fusidic acid [Verutex] belongs to the Bacterial protein synthesis inhibitor class. See all Bacterial protein synthesis inhibitor drugs at /class/bacterial-protein-synthesis-inhibitor.

What development phase is fusidic acid [Verutex] in?

fusidic acid [Verutex] is in Phase 3.

What are the side effects of fusidic acid [Verutex]?

Common side effects of fusidic acid [Verutex] include Gastrointestinal disturbances.

What does fusidic acid [Verutex] target?

fusidic acid [Verutex] targets Elongation factor G and is a Bacterial protein synthesis inhibitor.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing