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Fosmidomycin and Clindamycin co-administration

Jomaa Pharma GmbH · Phase 2 active Small molecule

Fosmidomycin inhibits the enzyme 1-deoxy-D-xylulose-5-phosphate reductoisomerase (DXR), which is involved in the biosynthesis of isoprenoids, while clindamycin inhibits protein synthesis by binding to the 50S ribosomal subunit.

Fosmidomycin inhibits the enzyme 1-deoxy-D-xylulose-5-phosphate reductoisomerase (DXR), which is involved in the biosynthesis of isoprenoids, while clindamycin inhibits protein synthesis by binding to the 50S ribosomal subunit. Used for Treatment of malaria caused by Plasmodium falciparum, Treatment of bacterial infections, including those caused by Streptococcus and Staphylococcus species.

At a glance

Generic nameFosmidomycin and Clindamycin co-administration
SponsorJomaa Pharma GmbH
Drug classAntibiotic
TargetDXR, 50S ribosomal subunit
ModalitySmall molecule
Therapeutic areaInfectious Diseases
PhasePhase 2

Mechanism of action

Fosmidomycin targets the DXR enzyme, which is crucial for the production of isoprenoids, a class of molecules involved in various cellular processes. By inhibiting DXR, fosmidomycin disrupts the isoprenoid biosynthesis pathway. Clindamycin, on the other hand, works by binding to the 50S ribosomal subunit, which prevents the elongation of the polypeptide chain during protein synthesis.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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