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fenofibrate + rosuvastatin

University of Campania Luigi Vanvitelli · FDA-approved active Small molecule

Fenofibrate activates PPAR-alpha to reduce triglycerides and increase HDL, while rosuvastatin inhibits HMG-CoA reductase to lower LDL cholesterol.

Fenofibrate activates PPAR-alpha to reduce triglycerides and increase HDL, while rosuvastatin inhibits HMG-CoA reductase to lower LDL cholesterol. Used for Dyslipidemia with elevated triglycerides and LDL cholesterol, Cardiovascular risk reduction in patients with mixed dyslipidemia.

At a glance

Generic namefenofibrate + rosuvastatin
SponsorUniversity of Campania Luigi Vanvitelli
Drug classFibrate + HMG-CoA reductase inhibitor (statin)
TargetPPAR-α; HMG-CoA reductase
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Fenofibrate is a fibrate that acts as a peroxisome proliferator-activated receptor alpha (PPAR-α) agonist, increasing lipolysis and reducing triglyceride-rich lipoprotein production while raising HDL cholesterol. Rosuvastatin is a statin that competitively inhibits HMG-CoA reductase, the rate-limiting enzyme in cholesterol synthesis, thereby reducing LDL cholesterol levels. Together, this combination addresses multiple lipid abnormalities in dyslipidemia.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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