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Fazirsiran Injection

Takeda · Phase 3 active Small molecule

Fazirsiran is an RNA interference (RNAi) therapeutic that silences the ALAS2 gene to reduce heme synthesis and iron accumulation in patients with iron overload disorders.

Fazirsiran is an RNA interference (RNAi) therapeutic that silences the ALAS2 gene to reduce heme synthesis and iron accumulation in patients with iron overload disorders. Used for Transfusion-dependent beta-thalassemia, Iron overload disorders.

At a glance

Generic nameFazirsiran Injection
Also known asTAK-999, ARO-AAT, ADS-001, TAK-999, ARO-AAT, ADS-001
SponsorTakeda
Drug classRNAi therapeutic (siRNA)
TargetALAS2 (aminolevulinic acid synthase 2)
ModalitySmall molecule
Therapeutic areaHematology / Rare Genetic Disorders
PhasePhase 3

Mechanism of action

Fazirsiran uses small interfering RNA (siRNA) technology to target and degrade ALAS2 messenger RNA, thereby decreasing aminolevulinic acid synthase 2 expression. This reduces heme production, which in turn decreases iron absorption and accumulation in tissues. The mechanism is designed to address the underlying pathophysiology of iron overload conditions by modulating the heme synthesis pathway.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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