Last reviewed · How we verify

Experimental: Aspirin monotherapy

Chinese Academy of Medical Sciences, Fuwai Hospital · FDA-approved active Small molecule

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes to reduce platelet aggregation and inflammation.

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes to reduce platelet aggregation and inflammation. Used for Secondary prevention of cardiovascular events (myocardial infarction, stroke), Primary prevention of cardiovascular disease in high-risk patients.

At a glance

Generic nameExperimental: Aspirin monotherapy
SponsorChinese Academy of Medical Sciences, Fuwai Hospital
Drug classNonsteroidal anti-inflammatory drug (NSAID) / Antiplatelet agent
TargetCOX-1, COX-2
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2, preventing the synthesis of thromboxane A2 and prostaglandins. This dual action reduces platelet clumping and inflammatory mediator production, making it effective for cardiovascular protection and pain relief. As monotherapy in this context, it is used as a standalone antiplatelet agent rather than in combination regimens.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results