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Idhifa (ENASIDENIB)

Bristol-Myers Squibb · FDA-approved approved Small molecule Quality 59/100

Idhifa works by blocking the IDH2 enzyme, which is mutated in some AML cells, to prevent the production of a toxic metabolite that promotes cancer growth.

Idhifa (enasidenib) is a small molecule inhibitor of isocitrate dehydrogenase 2 (IDH2), a mitochondrial enzyme. It was originally developed by Celgene Corp and is now owned by Bristol Myers Squibb. Idhifa is FDA-approved for the treatment of acute myeloid leukemia (AML) with a specific IDH2 mutation. The drug has a half-life of 7.9 days and is not yet off-patent. Key safety considerations include the risk of differentiation syndrome and IDH differentiation syndrome.

At a glance

Generic nameENASIDENIB
SponsorBristol-Myers Squibb
Drug classIsocitrate Dehydrogenase 2 Inhibitor
TargetIsocitrate dehydrogenase [NADP], mitochondrial
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved
First approval2017

Mechanism of action

Enasidenib is small molecule inhibitor of the isocitrate dehydrogenase (IDH2) enzyme. Enasidenib targets the mutant IDH2 variants R140Q, R172S, and R172K at approximately 40-fold lower concentrations than the wild-type enzyme in vitro. Inhibition of the mutant IDH2 enzyme by enasidenib led to decreased 2-hydroxyglutarate (2-HG) levels and induced myeloid differentiation in vitro and in vivo in mouse xenograft models of IDH2 mutated AML. In blood samples from patients with AML with mutated IDH2, enasidenib decreased 2-HG levels, reduced blast counts and increased percentages of mature myeloid cells.

Approved indications

Boxed warnings

Common side effects

Key clinical trials

Patents

PatentExpiryType

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results
FDA Orange BookPatents + exclusivity

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