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Dupixent (DUPILUMAB)

Regeneron · FDA-approved approved Monoclonal antibody Quality 62/100

Dupixent blocks the interleukin-4 receptor, a key player in the immune system's inflammatory response.

Dupixent (Dupilumab) is a biologic medication developed by Regeneron Pharmaceuticals, targeting the interleukin-4 receptor subunit alpha. It works as an interleukin-4 receptor alpha antagonist, modulating the immune system's response to reduce inflammation. Dupixent is approved to treat various conditions, including asthma, atopic dermatitis, chronic sinusitis, eosinophilic esophagitis, and prurigo nodularis. The medication is patented and commercially available, with key safety considerations including potential increased risk of infections and allergic reactions. As a biologic medication, Dupixent is not a small molecule.

At a glance

Generic nameDUPILUMAB
SponsorRegeneron
Drug classInterleukin-4 Receptor alpha Antagonist [EPC]
TargetInterleukin-4 receptor subunit alpha
ModalityMonoclonal antibody
Therapeutic areaImmunology
PhaseFDA-approved
First approval2017
Annual revenue14600

Mechanism of action

Dupilumab is human monoclonal IgG4 antibody that inhibits interleukin-4 (IL-4) and interleukin-13 (IL-13) signaling by specifically binding to the IL-4R subunit shared by the IL-4 and IL-13 receptor complexes. Dupilumab inhibits IL-4 signaling via the Type receptor and both IL-4 and IL-13 signaling through the Type II receptor.Inflammation driven by IL-4 and IL-13 is an important component in the pathogenesis of asthma, atopic dermatitis, and CRSwNP. Multiple cell types that express IL-4R (e.g., mast cells, eosinophils, macrophages, lymphocytes, epithelial cells, goblet cells) and inflammatory mediators (e.g., histamine, eicosanoids, leukotrienes, cytokines, chemokines) are involved in inflammation. Blocking IL-4R with dupilumab inhibits IL-4 and IL-13 cytokine-induced inflammatory responses, including the release of proinflammatory cytokines, chemokines, nitric oxide, and IgE. The mechanism of dupilumab action in asthma has not been definitively established.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results
SEC EDGARRevenue + earnings

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