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docetaxel, cisplatin, and capecitabine

Sun Yat-sen University · Phase 3 active Small molecule

Docetaxel inhibits microtubule dynamics, disrupting cell division, while cisplatin induces DNA crosslinks, causing cell death. Capecitabine is a prodrug of 5-fluorouracil, which inhibits thymidylate synthase, disrupting DNA synthesis.

Docetaxel inhibits microtubule dynamics, disrupting cell division, while cisplatin induces DNA crosslinks, causing cell death. Capecitabine is a prodrug of 5-fluorouracil, which inhibits thymidylate synthase, disrupting DNA synthesis. Used for Metastatic breast cancer, Metastatic non-small cell lung cancer, Adjuvant treatment of node-positive breast cancer.

At a glance

Generic namedocetaxel, cisplatin, and capecitabine
Also known asTPX induction chemotherapy
SponsorSun Yat-sen University
Drug classTaxane, Platinum-based chemotherapeutic, Antimetabolite
TargetMicrotubules, DNA
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Docetaxel binds to tubulin, preventing microtubule polymerization and causing cell cycle arrest at the G2/M phase. Cisplatin forms platinum-DNA adducts, triggering apoptosis. Capecitabine is converted to 5-fluorouracil, which inhibits thymidylate synthase, leading to DNA damage and cell death.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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