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Docetaxel, Cisplatin, 5-FU and Cetuximab

Hong Kong Nasopharyngeal Cancer Study Group Limited · Phase 2 active Small molecule

Docetaxel inhibits microtubule dynamics, Cisplatin induces DNA crosslinks, 5-FU inhibits thymidylate synthase, and Cetuximab binds to the epidermal growth factor receptor.

Docetaxel inhibits microtubule dynamics, Cisplatin induces DNA crosslinks, 5-FU inhibits thymidylate synthase, and Cetuximab binds to the epidermal growth factor receptor. Used for Locally advanced or metastatic non-small cell lung cancer, Locally advanced or metastatic breast cancer, Colorectal cancer, Head and neck cancer, Metastatic nasopharyngeal carcinoma.

At a glance

Generic nameDocetaxel, Cisplatin, 5-FU and Cetuximab
SponsorHong Kong Nasopharyngeal Cancer Study Group Limited
Drug classTaxane, Platinum-based chemotherapeutic, Antimetabolite, Monoclonal antibody
TargetMicrotubules, DNA, Thymidylate synthase, Epidermal growth factor receptor
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 2

Mechanism of action

Docetaxel works by stabilizing microtubules, preventing cell division, and ultimately leading to cell death. Cisplatin forms crosslinks in DNA, causing DNA damage and triggering cell death. 5-FU inhibits thymidylate synthase, an enzyme necessary for DNA synthesis, thereby preventing cancer cell growth. Cetuximab is a monoclonal antibody that binds to the epidermal growth factor receptor (EGFR), preventing its activation and subsequent signaling pathways that promote cell proliferation.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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