Last reviewed · How we verify

Discontinue Clopidogrel

Icahn School of Medicine at Mount Sinai · Phase 2 active Small molecule

Clopidogrel is a prodrug that is metabolized to its active form by CYP450 enzymes, primarily CYP2C19, which irreversibly inhibits platelet aggregation by blocking the P2Y12 receptor on platelets.

Clopidogrel is a prodrug that is metabolized to its active form by CYP450 enzymes, primarily CYP2C19, which irreversibly inhibits platelet aggregation by blocking the P2Y12 receptor on platelets. Used for Prevention of atherothrombotic events in patients with recent myocardial infarction, recent stroke, or established peripheral arterial disease.

At a glance

Generic nameDiscontinue Clopidogrel
SponsorIcahn School of Medicine at Mount Sinai
Drug classantiplatelet
TargetP2Y12 receptor
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 2

Mechanism of action

Clopidogrel acts as an antiplatelet agent by selectively inhibiting the P2Y12 receptor, which is involved in the ADP-mediated activation of platelets. This inhibition prevents the amplification of the platelet activation process and reduces the formation of thrombi.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results