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Direct oral anticoagulant

Centre Hospitalier Universitaire de Nīmes · Phase 3 active Small molecule

Direct oral anticoagulants inhibit specific clotting factors to prevent blood clot formation.

Direct oral anticoagulants inhibit specific clotting factors to prevent blood clot formation. Used for Atrial fibrillation for stroke prevention, Venous thromboembolism (deep vein thrombosis and pulmonary embolism) treatment and prevention, Acute coronary syndrome (in combination with antiplatelet therapy).

At a glance

Generic nameDirect oral anticoagulant
Also known asDOAC, low molecular weight heparin
SponsorCentre Hospitalier Universitaire de Nīmes
Drug classDirect oral anticoagulant (DOAC)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

Direct oral anticoagulants (DOACs) work by directly inhibiting either Factor Xa or thrombin (Factor IIa) in the coagulation cascade, preventing the formation of fibrin clots. This class includes Factor Xa inhibitors (apixaban, rivaroxaban, edoxaban) and direct thrombin inhibitors (dabigatran). By blocking these key enzymes, DOACs reduce thrombotic events while maintaining hemostasis.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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