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Dexamethasone (intravenous)

Poznan University of Medical Sciences · Phase 3 active Small molecule Under review

Dexamethasone (intravenous) is a Glucocorticoid (corticosteroid) Small molecule drug developed by Poznan University of Medical Sciences. It is currently in Phase 3 development for Cerebral edema associated with primary or metastatic brain tumors, Severe inflammatory or allergic conditions, Acute respiratory distress syndrome (ARDS).

Dexamethasone is a synthetic glucocorticoid that suppresses the immune system and reduces inflammation by binding to glucocorticoid receptors in the cytoplasm.

Dexamethasone, a small molecule, is used intravenously to treat various conditions, including Arthropathy of Knee, Hip Dysplasia, Postoperative Complications, Shoulder Injury, and Relapsing Multiple Sclerosis. It is administered in the form of Dexamethasone Sodium Phosphate or Dexamethasone itself, often as part of a clinical trial or study.

Likelihood of approval
61.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Oncology Phase 3 boost +3.0pp
    Oncology Phase 3 trials have higher approval rates (~61%) than the cross-industry average due to clearer endpoints and FDA oncology pathway.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameDexamethasone (intravenous)
SponsorPoznan University of Medical Sciences
Drug classGlucocorticoid (corticosteroid)
TargetGlucocorticoid receptor (GR)
ModalitySmall molecule
Therapeutic areaImmunology, Inflammation, Oncology (supportive care)
PhasePhase 3

Mechanism of action

Dexamethasone enters cells and binds to intracellular glucocorticoid receptors, which then translocate to the nucleus and modulate gene transcription. This leads to decreased production of pro-inflammatory cytokines, reduced immune cell activation and proliferation, and suppression of inflammatory mediators. The intravenous formulation provides rapid systemic delivery for acute inflammatory or immunological conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about Dexamethasone (intravenous)

What is Dexamethasone (intravenous)?

Dexamethasone (intravenous) is a Glucocorticoid (corticosteroid) drug developed by Poznan University of Medical Sciences, indicated for Cerebral edema associated with primary or metastatic brain tumors, Severe inflammatory or allergic conditions, Acute respiratory distress syndrome (ARDS).

How does Dexamethasone (intravenous) work?

Dexamethasone is a synthetic glucocorticoid that suppresses the immune system and reduces inflammation by binding to glucocorticoid receptors in the cytoplasm.

What is Dexamethasone (intravenous) used for?

Dexamethasone (intravenous) is indicated for Cerebral edema associated with primary or metastatic brain tumors, Severe inflammatory or allergic conditions, Acute respiratory distress syndrome (ARDS), Septic shock.

Who makes Dexamethasone (intravenous)?

Dexamethasone (intravenous) is developed by Poznan University of Medical Sciences (see full Poznan University of Medical Sciences pipeline at /company/poznan-university-of-medical-sciences).

What drug class is Dexamethasone (intravenous) in?

Dexamethasone (intravenous) belongs to the Glucocorticoid (corticosteroid) class. See all Glucocorticoid (corticosteroid) drugs at /class/glucocorticoid-corticosteroid.

What development phase is Dexamethasone (intravenous) in?

Dexamethasone (intravenous) is in Phase 3.

What are the side effects of Dexamethasone (intravenous)?

Common side effects of Dexamethasone (intravenous) include Hyperglycemia, Hypertension, Insomnia, Mood changes (agitation, anxiety), Immunosuppression / increased infection risk, Hypokalemia.

What does Dexamethasone (intravenous) target?

Dexamethasone (intravenous) targets Glucocorticoid receptor (GR) and is a Glucocorticoid (corticosteroid).

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing