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Denosumab (Prolia)

Prince of Wales Hospital, Shatin, Hong Kong · FDA-approved active Small molecule

Denosumab (Prolia) is a RANKL inhibitor (monoclonal antibody) Small molecule drug developed by Prince of Wales Hospital, Shatin, Hong Kong. It is currently FDA-approved for Postmenopausal osteoporosis, Bone metastases from solid tumors, Giant cell tumor of bone. Also known as: Prolia, Xgeva.

Denosumab is a monoclonal antibody that binds to RANKL, preventing bone resorption by inhibiting osteoclast formation and activation.

Denosumab is a monoclonal antibody that binds to RANKL, preventing bone resorption by inhibiting osteoclast formation and activation. Used for Postmenopausal osteoporosis, Bone metastases from solid tumors, Giant cell tumor of bone.

At a glance

Generic nameDenosumab (Prolia)
Also known asProlia, Xgeva
SponsorPrince of Wales Hospital, Shatin, Hong Kong
Drug classRANKL inhibitor (monoclonal antibody)
TargetRANKL (receptor activator of nuclear factor kappa-B ligand)
ModalitySmall molecule
Therapeutic areaBone metabolism / Oncology
PhaseFDA-approved

Mechanism of action

Denosumab targets RANKL (receptor activator of nuclear factor kappa-B ligand), a key mediator of osteoclast differentiation and survival. By blocking RANKL signaling, it reduces bone turnover and increases bone mineral density. This mechanism makes it effective for treating conditions characterized by excessive bone loss, such as osteoporosis and bone metastases.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

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Frequently asked questions about Denosumab (Prolia)

What is Denosumab (Prolia)?

Denosumab (Prolia) is a RANKL inhibitor (monoclonal antibody) drug developed by Prince of Wales Hospital, Shatin, Hong Kong, indicated for Postmenopausal osteoporosis, Bone metastases from solid tumors, Giant cell tumor of bone.

How does Denosumab (Prolia) work?

Denosumab is a monoclonal antibody that binds to RANKL, preventing bone resorption by inhibiting osteoclast formation and activation.

What is Denosumab (Prolia) used for?

Denosumab (Prolia) is indicated for Postmenopausal osteoporosis, Bone metastases from solid tumors, Giant cell tumor of bone, Hypercalcemia of malignancy.

Who makes Denosumab (Prolia)?

Denosumab (Prolia) is developed and marketed by Prince of Wales Hospital, Shatin, Hong Kong (see full Prince of Wales Hospital, Shatin, Hong Kong pipeline at /company/prince-of-wales-hospital-shatin-hong-kong).

Is Denosumab (Prolia) also known as anything else?

Denosumab (Prolia) is also known as Prolia, Xgeva.

What drug class is Denosumab (Prolia) in?

Denosumab (Prolia) belongs to the RANKL inhibitor (monoclonal antibody) class. See all RANKL inhibitor (monoclonal antibody) drugs at /class/rankl-inhibitor-monoclonal-antibody.

What development phase is Denosumab (Prolia) in?

Denosumab (Prolia) is FDA-approved (marketed).

What are the side effects of Denosumab (Prolia)?

Common side effects of Denosumab (Prolia) include Hypocalcemia, Osteonecrosis of the jaw, Atypical femoral fractures, Back pain, Arthralgia, Infection.

What does Denosumab (Prolia) target?

Denosumab (Prolia) targets RANKL (receptor activator of nuclear factor kappa-B ligand) and is a RANKL inhibitor (monoclonal antibody).

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing