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DCV/ASV/BMS-791325

Bristol-Myers Squibb · Phase 3 active Small molecule ✓ Verified May 2026

DCV/ASV/BMS-791325 is a Direct-acting antiviral combination (NS5A inhibitor + NS3 protease inhibitor + NS5B polymerase inhibitor) Small molecule drug developed by Bristol-Myers Squibb. It is currently in Phase 3 development for Chronic hepatitis C virus infection (genotype 1-6). Also known as: Fixed Dose Combination (FDC) of Daclatasvir/Asunaprevir/BMS-791325.

This is a three-drug combination of direct-acting antivirals (DCV, ASV, and BMS-791325) that inhibit hepatitis C virus NS5A, NS3 protease, and NS5B polymerase respectively to block viral replication.

DCV/ASV/BMS-791325 is a small molecule combination treatment that has been studied in clinical trials for Hepatitis C Infection, Hepatitis C Virus Genotype 4 Infection, and Hepatitis C. This combination treatment has been investigated in a study on its pharmacokinetic effects in healthy volunteers, specifically in relation to the selective serotonin reuptake inhibitors escitalopram and sertraline.

Likelihood of approval
61.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Big-pharma sponsor +3.0pp
    Bristol-Myers Squibb is a top-20 pharma sponsor — historical approval rates run ~3pp above average due to scale, regulatory experience, and trial-design quality.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameDCV/ASV/BMS-791325
Also known asFixed Dose Combination (FDC) of Daclatasvir/Asunaprevir/BMS-791325
SponsorBristol-Myers Squibb
Drug classDirect-acting antiviral combination (NS5A inhibitor + NS3 protease inhibitor + NS5B polymerase inhibitor)
TargetHCV NS5A, NS3/4A protease, NS5B polymerase
ModalitySmall molecule
Therapeutic areaVirology / Hepatology
PhasePhase 3

Mechanism of action

DCV (daclatasvir) inhibits the NS5A protein, ASV (asunaprevir) inhibits the NS3/4A serine protease, and BMS-791325 inhibits the NS5B RNA-dependent RNA polymerase. Together, these three agents target multiple steps of the hepatitis C viral lifecycle, preventing viral replication and clearance of infection. This pan-genotypic combination was designed to achieve high cure rates across different HCV genotypes with a short treatment duration.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about DCV/ASV/BMS-791325

What is DCV/ASV/BMS-791325?

DCV/ASV/BMS-791325 is a Direct-acting antiviral combination (NS5A inhibitor + NS3 protease inhibitor + NS5B polymerase inhibitor) drug developed by Bristol-Myers Squibb, indicated for Chronic hepatitis C virus infection (genotype 1-6).

How does DCV/ASV/BMS-791325 work?

This is a three-drug combination of direct-acting antivirals (DCV, ASV, and BMS-791325) that inhibit hepatitis C virus NS5A, NS3 protease, and NS5B polymerase respectively to block viral replication.

What is DCV/ASV/BMS-791325 used for?

DCV/ASV/BMS-791325 is indicated for Chronic hepatitis C virus infection (genotype 1-6).

Who makes DCV/ASV/BMS-791325?

DCV/ASV/BMS-791325 is developed by Bristol-Myers Squibb (see full Bristol-Myers Squibb pipeline at /company/bristol-myers-squibb).

Is DCV/ASV/BMS-791325 also known as anything else?

DCV/ASV/BMS-791325 is also known as Fixed Dose Combination (FDC) of Daclatasvir/Asunaprevir/BMS-791325.

What drug class is DCV/ASV/BMS-791325 in?

DCV/ASV/BMS-791325 belongs to the Direct-acting antiviral combination (NS5A inhibitor + NS3 protease inhibitor + NS5B polymerase inhibitor) class. See all Direct-acting antiviral combination (NS5A inhibitor + NS3 protease inhibitor + NS5B polymerase inhibitor) drugs at /class/direct-acting-antiviral-combination-ns5a-inhibitor-ns3-protease-inhibitor-ns5b-polymerase-inhibitor.

What development phase is DCV/ASV/BMS-791325 in?

DCV/ASV/BMS-791325 is in Phase 3.

What are the side effects of DCV/ASV/BMS-791325?

Common side effects of DCV/ASV/BMS-791325 include Headache, Fatigue, Nausea, Diarrhea.

What does DCV/ASV/BMS-791325 target?

DCV/ASV/BMS-791325 targets HCV NS5A, NS3/4A protease, NS5B polymerase and is a Direct-acting antiviral combination (NS5A inhibitor + NS3 protease inhibitor + NS5B polymerase inhibitor).

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing