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Dapagliflozin (Forxiga)

National Institute of Cardiology, Warsaw, Poland · FDA-approved active Small molecule

Dapagliflozin inhibits sodium-glucose cotransporter 2 (SGLT2) in the kidney, reducing glucose reabsorption and increasing urinary glucose excretion.

Dapagliflozin inhibits sodium-glucose cotransporter 2 (SGLT2) in the kidney, reducing glucose reabsorption and increasing urinary glucose excretion. Used for Type 2 diabetes mellitus, Heart failure with reduced ejection fraction, Chronic kidney disease.

At a glance

Generic nameDapagliflozin (Forxiga)
Also known asSGLT-2 inhibitor, Farxiga
SponsorNational Institute of Cardiology, Warsaw, Poland
Drug classSGLT2 inhibitor
TargetSGLT2 (sodium-glucose cotransporter 2)
ModalitySmall molecule
Therapeutic areaDiabetes, Cardiovascular
PhaseFDA-approved

Mechanism of action

By blocking SGLT2, dapagliflozin prevents the reabsorption of filtered glucose in the proximal tubule of the nephron, leading to increased urinary glucose loss and lower blood glucose levels. Beyond glycemic control, SGLT2 inhibition reduces cardiac preload and afterload, improves myocardial energetics, and has cardioprotective and renoprotective effects independent of glucose lowering.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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