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Cyclosporine Oral Product

Peking University People's Hospital · FDA-approved active Small molecule

Cyclosporine suppresses T-cell activation and proliferation by inhibiting calcineurin, thereby reducing the production of pro-inflammatory cytokines.

Cyclosporine suppresses T-cell activation and proliferation by inhibiting calcineurin, thereby reducing the production of pro-inflammatory cytokines. Used for Prevention of organ rejection in transplant recipients, Autoimmune diseases including rheumatoid arthritis and nephrotic syndrome, Severe atopic dermatitis.

At a glance

Generic nameCyclosporine Oral Product
Also known asPrednisone, Sandimmune, cyclosporine A
SponsorPeking University People's Hospital
Drug classCalcineurin inhibitor
TargetCalcineurin (protein phosphatase 2B)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Cyclosporine binds to cyclophilin and the resulting complex inhibits calcineurin phosphatase, which prevents dephosphorylation and nuclear translocation of NFAT (nuclear factor of activated T cells). This blocks the transcription of IL-2 and other cytokines essential for T-cell activation and proliferation, leading to immunosuppression. It is used to prevent organ rejection and treat autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results