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Cyclosporine (Neoral)

Novartis · FDA-approved active Small molecule

Cyclosporine inhibits T-cell activation by binding to cyclophilin and blocking calcineurin-mediated transcription of interleukin-2 and other cytokines.

Cyclosporine inhibits T-cell activation by binding to cyclophilin and blocking calcineurin-mediated transcription of interleukin-2 and other cytokines. Used for Organ transplant rejection prophylaxis (kidney, heart, liver, pancreas), Severe active rheumatoid arthritis, Severe psoriasis.

At a glance

Generic nameCyclosporine (Neoral)
Also known asNeoral
SponsorNovartis
Drug classCalcineurin inhibitor
TargetCalcineurin (via cyclophilin binding)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Cyclosporine forms a complex with the intracellular protein cyclophilin, which then inhibits the phosphatase calcineurin. This prevents dephosphorylation and nuclear translocation of NFAT (nuclear factor of activated T cells), thereby suppressing the transcription of IL-2 and other pro-inflammatory cytokines essential for T-cell proliferation and activation. The result is potent immunosuppression without direct cytotoxicity.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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