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Selg1 (CRIZANLIZUMAB)

Novartis · FDA-approved withdrawn Monoclonal antibody Quality 48/100

Selg1 works by binding to P-selectin, preventing it from interacting with its ligands.

Selg1 (Crizanlizumab) is a selectin blocker developed by Novartis Pharmaceuticals Corp, targeting P-selectin to treat vaso-occlusive crisis in sickle cell disease. It was FDA-approved in 2019 and remains a patented product. As a small molecule, it works by binding to P-selectin, preventing it from interacting with its ligands and thereby reducing the frequency of vaso-occlusive crises. Key safety considerations include the potential for infusion reactions and thrombocytopenia. Selg1 is a valuable treatment option for patients with sickle cell disease.

At a glance

Generic nameCRIZANLIZUMAB
SponsorNovartis
Drug classSelectin Blocker [EPC]
TargetP-selectin
ModalityMonoclonal antibody
Therapeutic areaMetabolic
PhaseFDA-approved
First approval2019

Mechanism of action

Crizanlizumab-tmca is humanized IgG2 kappa monoclonal antibody that binds to P-selectin and blocks interactions with its ligands, including P-selectin glycoprotein ligand (PSGL-1). Crizanlizumab-tmca can also dissociate preformed P-selectin/PSGL-1 complex. Binding P-selectin on the surface of the activated endothelium and platelets blocks interactions between endothelial cells, platelets, red blood cells, and leukocytes.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results

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