Last reviewed · How we verify

Conventional Antiarrhythmic Drug Therapy

Population Health Research Institute · Phase 3 active Small molecule

Conventional antiarrhythmic drugs work by modulating cardiac electrical activity through various mechanisms including sodium channel blockade, beta-adrenergic antagonism, potassium channel inhibition, or calcium channel blockade to restore normal heart rhythm.

Conventional antiarrhythmic drugs work by modulating cardiac electrical activity through various mechanisms including sodium channel blockade, beta-adrenergic antagonism, potassium channel inhibition, or calcium channel blockade to restore normal heart rhythm. Used for Atrial fibrillation management, Ventricular arrhythmias, Supraventricular tachycardia.

At a glance

Generic nameConventional Antiarrhythmic Drug Therapy
SponsorPopulation Health Research Institute
Drug classAntiarrhythmic agent (mixed class)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

Conventional antiarrhythmic drugs are classified into four main groups (Vaughan-Williams classification) that target different aspects of cardiac electrophysiology. Class I agents block sodium channels to slow conduction, Class II agents are beta-blockers that reduce automaticity, Class III agents block potassium channels to prolong repolarization, and Class IV agents block calcium channels to slow AV nodal conduction. These mechanisms collectively suppress ectopic activity and restore normal sinus rhythm in patients with various arrhythmias.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results