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conbercept, Fixed

Chengdu Kanghong Biotech Co., Ltd. · Phase 3 active Small molecule Under review Quality 0/100

conbercept, Fixed is a VEGF decoy receptor Small molecule drug developed by Chengdu Kanghong Biotech Co., Ltd.. It is currently in Phase 3 development for Diabetic macular edema.

Conbercept is a recombinant human VEGF decoy receptor that inhibits angiogenesis.

Conbercept is a protein-based inhibitor of vascular endothelial growth factor A, used to treat conditions such as choroidal neovascularization secondary to degenerative myopia and diabetic macular edema. It is administered via injection, with fixed and PRN (as needed) dosing regimens available.

Likelihood of approval
58.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameconbercept, Fixed
SponsorChengdu Kanghong Biotech Co., Ltd.
Drug classVEGF decoy receptor
TargetVEGF
ModalitySmall molecule
Therapeutic areaOphthalmology
PhasePhase 3

Mechanism of action

Conbercept works by binding to vascular endothelial growth factor (VEGF) and preventing it from interacting with its receptors on the surface of endothelial cells, thereby inhibiting angiogenesis. This mechanism of action is thought to be responsible for its anti-tumor and anti-angiogenic effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about conbercept, Fixed

What is conbercept, Fixed?

conbercept, Fixed is a VEGF decoy receptor drug developed by Chengdu Kanghong Biotech Co., Ltd., indicated for Diabetic macular edema.

How does conbercept, Fixed work?

Conbercept is a recombinant human VEGF decoy receptor that inhibits angiogenesis.

What is conbercept, Fixed used for?

conbercept, Fixed is indicated for Diabetic macular edema.

Who makes conbercept, Fixed?

conbercept, Fixed is developed by Chengdu Kanghong Biotech Co., Ltd. (see full Chengdu Kanghong Biotech Co., Ltd. pipeline at /company/chengdu-kanghong-biotech-co-ltd).

What drug class is conbercept, Fixed in?

conbercept, Fixed belongs to the VEGF decoy receptor class. See all VEGF decoy receptor drugs at /class/vegf-decoy-receptor.

What development phase is conbercept, Fixed in?

conbercept, Fixed is in Phase 3.

What are the side effects of conbercept, Fixed?

Common side effects of conbercept, Fixed include conjunctival hemorrhage, eye pain, conjunctival hyperemia.

What does conbercept, Fixed target?

conbercept, Fixed targets VEGF and is a VEGF decoy receptor.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing