Last reviewed · How we verify

Colchicine only

The George Institute · Phase 3 active Small molecule

Colchicine inhibits microtubule polymerization by binding to tubulin, thereby suppressing inflammatory cell migration and activation.

Colchicine inhibits microtubule polymerization by binding to tubulin, thereby suppressing inflammatory cell migration and activation. Used for Acute gout flare prophylaxis and treatment, Familial Mediterranean fever, Cardiovascular disease (post-myocardial infarction, atherosclerosis) — Phase 3 investigation.

At a glance

Generic nameColchicine only
SponsorThe George Institute
Drug classMicrotubule inhibitor / Anti-inflammatory agent
Targetβ-tubulin
ModalitySmall molecule
Therapeutic areaCardiovascular / Immunology / Rheumatology
PhasePhase 3

Mechanism of action

Colchicine binds to β-tubulin and prevents microtubule assembly, which disrupts cell division and inflammatory cell movement. This leads to reduced recruitment of neutrophils and other immune cells to sites of inflammation, and decreases the production of inflammatory mediators like IL-1β and TNF-α. The drug has been used for decades in gout and is now being investigated for cardiovascular and other inflammatory indications.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results