Last reviewed · How we verify

CNI (≥75%)

Hoffmann-La Roche · FDA-approved active Small molecule

CNI (calcineurin inhibitor) suppresses T-cell activation by inhibiting calcineurin phosphatase, thereby blocking IL-2 production and T-cell proliferation.

CNI (calcineurin inhibitor) suppresses T-cell activation by inhibiting calcineurin phosphatase, thereby blocking IL-2 production and T-cell proliferation. Used for Organ transplant rejection prophylaxis (renal, cardiac, hepatic), Autoimmune diseases (atopic dermatitis, psoriasis, rheumatoid arthritis).

At a glance

Generic nameCNI (≥75%)
SponsorHoffmann-La Roche
Drug classCalcineurin inhibitor
TargetCalcineurin (protein phosphatase 2B)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Calcineurin inhibitors bind to immunophilins (cyclophilin or FK-binding protein) and form a complex that inhibits calcineurin, a serine/threonine phosphatase essential for dephosphorylating NFAT transcription factors. This prevents NFAT nuclear translocation and subsequent IL-2 and other cytokine gene transcription, resulting in suppression of T-cell activation and proliferation. CNIs are used primarily as immunosuppressants in transplantation and autoimmune diseases.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results