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CNI (50%)

Hoffmann-La Roche · FDA-approved active Small molecule

CNI (calcineurin inhibitor) suppresses T-cell activation by inhibiting calcineurin phosphatase, thereby reducing production of pro-inflammatory cytokines.

CNI (calcineurin inhibitor) suppresses T-cell activation by inhibiting calcineurin phosphatase, thereby reducing production of pro-inflammatory cytokines. Used for Organ transplant rejection prophylaxis, Autoimmune and inflammatory conditions (e.g., atopic dermatitis, psoriasis).

At a glance

Generic nameCNI (50%)
SponsorHoffmann-La Roche
Drug classCalcineurin inhibitor
TargetCalcineurin (protein phosphatase 2B)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Calcineurin inhibitors bind to immunophilins (cyclophilin or FK-binding protein) and block calcineurin-mediated dephosphorylation of NFAT, a transcription factor essential for IL-2 and other cytokine gene expression in T cells. This prevents T-cell proliferation and differentiation, making them potent immunosuppressants used in transplantation and autoimmune diseases.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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