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Clostridium Botulinum Toxin Type A

Daewoong Pharmaceutical Co. LTD. · FDA-approved active Small molecule

Clostridium botulinum toxin type A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis.

Clostridium botulinum toxin type A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis. Used for Cervical dystonia, Blepharospasm, Hemifacial spasm.

At a glance

Generic nameClostridium Botulinum Toxin Type A
Also known asNABOTA, BOTOX
SponsorDaewoong Pharmaceutical Co. LTD.
Drug classNeurotoxin; botulinum toxin
TargetSNAP-25 (synaptosome-associated protein of 25 kDa)
ModalitySmall molecule
Therapeutic areaNeurology; Aesthetics; Pain Management
PhaseFDA-approved

Mechanism of action

The toxin is a zinc-dependent endopeptidase that cleaves SNAP-25 (synaptosome-associated protein of 25 kDa), preventing the fusion of acetylcholine-containing vesicles with the presynaptic membrane. This results in temporary paralysis of muscles at the injection site, lasting 3-4 months before neuromuscular function gradually recovers through sprouting of new nerve terminals.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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