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NIRAPARIB

FDA-approved approved Small molecule Quality 48/100

Niraparib inhibits PARP enzymes, causing DNA damage and cell death, while abiraterone inhibits CYP17, reducing androgen production.

Niraparib is a marketed PARP inhibitor primarily indicated for BRCA2-mutated metastatic castration-sensitive prostate cancer (mCSPC). Its key strength lies in its mechanism of action, which effectively causes DNA damage and cell death in cancer cells, providing a targeted therapy option. The primary risk is the key composition patent expiry in 2028, which could lead to increased competition from generics.

At a glance

Generic nameNIRAPARIB
TargetPARP-1, PARP-2, CYP17
ModalitySmall molecule
PhaseFDA-approved
First approval2017

Mechanism of action

Niraparib works by blocking PARP enzymes, which are involved in DNA repair. This leads to DNA damage, apoptosis, and cell death, especially in cells with BRCA1/2 deficiencies. Abiraterone, on the other hand, inhibits the CYP17 enzyme, which is crucial for androgen production, thereby reducing androgen levels and slowing tumor growth in androgen-sensitive prostate cancer.

Approved indications

Common side effects

Drug interactions

Key clinical trials

Patents

PatentExpiryType

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results
FDA Orange BookPatents + exclusivity

Competitive intelligence

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