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Cetuximab + FOLFIRI

Cinnagen · Phase 3 active Small molecule

Cetuximab blocks EGFR signaling while FOLFIRI chemotherapy damages DNA, together targeting colorectal cancer cells through dual mechanisms.

Cetuximab blocks EGFR signaling while FOLFIRI chemotherapy damages DNA, together targeting colorectal cancer cells through dual mechanisms. Used for Metastatic colorectal cancer, KRAS wild-type, Recurrent or metastatic colorectal cancer as first-line or later-line therapy.

At a glance

Generic nameCetuximab + FOLFIRI
Also known asFOLFIRI = irinotecan + calcium folinate + 5-fluorouracil
SponsorCinnagen
Drug classMonoclonal antibody + chemotherapy combination
TargetEGFR (cetuximab component); Topoisomerase I (irinotecan component)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Cetuximab is a monoclonal antibody that binds to and inhibits the epidermal growth factor receptor (EGFR), blocking downstream proliferation signals in cancer cells. FOLFIRI is a chemotherapy regimen combining fluorouracil, leucovorin, and irinotecan that damages DNA and inhibits topoisomerase I. The combination leverages targeted immunotherapy with cytotoxic chemotherapy to enhance efficacy in EGFR-expressing colorectal cancers.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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