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CEDAZURIDINE

FDA-approved approved Small molecule Quality 45/100

Decitabine inhibits DNA methyltransferase, leading to DNA hypomethylation and cellular differentiation or apoptosis.

CEDAZURIDINE is a marketed drug indicated for Myelodysplastic Syndromes (MDS), leveraging decitabine's mechanism of inhibiting DNA methyltransferase to induce DNA hypomethylation and cellular differentiation or apoptosis. The drug's key strength lies in its unique mechanism of action, which differentiates it from other treatments in the MDS market. The primary risk is the key composition patent expiry in 2028, which could lead to increased competition from generics.

At a glance

Generic nameCEDAZURIDINE
Drug classNucleoside Metabolic Inhibitor [EPC]
TargetDNA methyltransferase
ModalitySmall molecule
PhaseFDA-approved
First approval2020

Mechanism of action

Decitabine works by being incorporated into DNA and inhibiting DNA methyltransferase, which reduces DNA methylation. This can restore normal gene function and lead to cell differentiation or death, particularly in rapidly dividing cancer cells.

Approved indications

Common side effects

Drug interactions

Key clinical trials

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results