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carbidopa-levodopa (Sinemet)

Baylor College of Medicine · FDA-approved active Small molecule

Carbidopa inhibits peripheral decarboxylation of levodopa, allowing more levodopa to reach the brain where it is converted to dopamine to replace depleted neurotransmitter in Parkinson's disease.

Carbidopa inhibits peripheral decarboxylation of levodopa, allowing more levodopa to reach the brain where it is converted to dopamine to replace depleted neurotransmitter in Parkinson's disease. Used for Parkinson's disease, Parkinsonism secondary to carbon monoxide poisoning or manganese intoxication.

At a glance

Generic namecarbidopa-levodopa (Sinemet)
SponsorBaylor College of Medicine
Drug classDopamine replacement therapy
TargetAromatic amino acid decarboxylase (AADC); dopamine pathway
ModalitySmall molecule
Therapeutic areaNeurology
PhaseFDA-approved

Mechanism of action

Levodopa is a dopamine precursor that crosses the blood-brain barrier, but is rapidly metabolized peripherally by aromatic amino acid decarboxylase (AADC). Carbidopa is a peripheral AADC inhibitor that does not cross the blood-brain barrier, thereby reducing peripheral conversion of levodopa to dopamine and increasing the fraction available for central nervous system delivery. This combination allows lower levodopa doses and reduces peripheral dopamine-related side effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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